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"The potential of new nicotine and tobacco products as tools for people who smoke to quit combustible cigarettes a systematic review of common practices and guidance towards a robust study protocol to measure cessation efficacy"

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Commensal biofilms in tobacco smoke environments faced a loss of viability and elicited an early proinflammatory immune response (seen as an increase in various interleukins and macrophage inflammatory proteins) from immortalised human oral keratinocytes (OKF6/TERT-2) [35]